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Anal sac carcinoma

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    Back to Fact Sheets. Download PDF. This is described as an uncommon tumour but we do seem to see a significant number of patients with this disease, no doubt partly due to this being a special interest in our clinic. In a number of cases this is the only site where the tumour is growing, but in other cases they may spread to the lymph nodes or via the bloodstream to places like the lungs, liver and spleen. Sometimes these tumours are associated with the production of a hormone that causes excessive drinking and urination. In some circumstances this may be the only problem evident prior to diagnosis of the tumour.
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    Metastatic anal sac carcinoma with hypercalcaemia and associated hypertrophic osteopathy in a dog

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    Metastatic anal sac carcinoma with hypercalcaemia and associated hypertrophic osteopathy in a dog

    Other treatment modalities include radiotherapy and chemotherapy, but the role of these treatments has not been clearly determined yet. Pre-operative medical support is provided as required. In particular, severe hypercalcaemia of malignancy HM; i. Management of HM is beyond the scope of this text and is widely detailed elsewhere Briefly, it is treated with a combination of fluid therapy 0. Surgery must aim at decreasing the tumour burden as much as possible, especially in hypercalcaemic patients.
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    Canine anal sac carcinomas – treatment and prognosis

    The tumour is invasive with a high mortality rate. The cancerous cells may invade regional lymph nodes and travel to the spine, lungs, liver and spleen via the bloodstream. The tumour may go unnoticed in the early stages and an abnormal mass or swelling in the region of the anus perineal area may only be recognised when the growth is large. Other signs of anal sac adenocarcinoma include problems with defecation, local pain or irritation, excessive drinking and urination, hind limb weakness and lethargy.
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    A seven-year-old male neutered Irish setter was treated for a metastatic anal sac adenocarcinoma ASAC and hypercalcaemia by complete surgical excision of the primary tumour and partial excision of the sublumbar lymph nodes. Further enlargement of the sublumbar lymph nodes was linked to recurrent hypercalcaemia 3 months after surgical treatment. Medical treatment with Toceranib and Clodronate showed modest results in the treatment of the tumour and the hypercalcaemia. Radiotherapy of the sublumbar lymph nodes and later concurrent carboplatin chemotherapy resulted in partial tumour remission with marked reduction in size of the lymph nodes and normalization of the calcaemia.
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